Tecar Currents Explained: For Clinicians and Curious Patients
Breakdown: Each Current Mode & Their Tissue Effects
1. Capacitive Mode (CET)
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Description / physics: The active electrode is insulated (dielectric), creating an alternating electric field. The underlying tissue with higher permittivity and fluid content becomes “polarized” and responds to the oscillations.
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Depth & distribution: More superficial heating, because the field is concentrated near tissues with higher water content (muscle, fat, skin).
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Tissues targeted: Soft tissues like muscle, vascular/lymphatic beds, subcutaneous tissues, superficial fascia, superficial tissues.
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Effects & healing mechanism:
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Increased perfusion / vasodilation: Heating drives local circulation, delivering nutrients & O₂, clearing metabolites.
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Increased cell membrane permeability / ionic exchange: The oscillating field helps drive nutrients and metabolites across cell membranes, facilitating repair.
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Thermal relaxation / muscle relaxation: Heat helps reduce stiffness and muscle guarding, allowing improved range of motion.
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Pain modulation: By warming tissues, reducing stiffness, and modulating nerve conduction, capacitive mode supports analgesia.
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2. Resistive Mode (RET)
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Description / physics: The active electrode is uninsulated (conductive), so the current preferentially travels through more resistive tissues—the path of least conduction is through denser, higher-impedance structures (bone, tendons, ligaments). winback.com+3winback
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Depth & distribution: More “deep heating” in higher resistance tissues (bones, joint capsules, tendon insertions).
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Tissues targeted: Tendons, ligaments, joint capsules, bone interfaces, denser connective tissue, deep fascia.
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Effects & healing mechanism:
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Localized heating in depth: In resistive mode, heating is concentrated in denser resistive tissues, more so in structures like tendon insertions or bone–tissue interfaces. For example, in phantom models, RET caused a temperature rise concentrated around a bone inclusion. winback-academy.org
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Stimulated remodeling in connective tissues: The heat stimulates fibroblasts and collagen reorganization, aiding tendon / ligament healing.
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Improved circulation in surrounding tissues: Even though RET favors deep resistive structures, the surrounding tissues also benefit via vascular response.
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Pain reduction & tissue extensibility: As stiffness in deep tissues relaxes, load tolerance improves.
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3. Mixed / Synchronous CET + RET (“combination” / dual modes)
Many devices (especially newer Winback models) let you apply capacitive and resistive simultaneously or rapidly alternate, allowing you to treat both soft tissues and deep connective tissues in tandem.
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Benefit: You don’t have to choose superficial vs deep—you can engage both layers in one therapeutic window.
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Mechanism synergy: Soft tissue warming supports vascular access and metabolic exchange, while deep heating supports connective tissue remodeling and restructuring.
4. Hi-EMS (medium-frequency motor stimulation)
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Description / physics: A modulated medium-frequency current (in the 1,500–4,000 Hz range) designed to activate motor nerves and induce muscle contractions without being uncomfortably strong.
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Tissues targeted: Motor nerve fibers (neuromuscular junctions), muscle fibers (both superficial and deep).
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Effects & healing mechanism:
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Muscle activation even when voluntary contraction is limited: Helps “pump” blood/lymph, enhance circulation, and prevent muscle atrophy during immobilization periods.
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Venous / lymphatic pumping effect: Repeated contractions help flush edema and metabolic waste.
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Neuromuscular re-education: Promotes proper recruitment patterns and combats inhibition after pain or injury.
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Synergy with thermal effects: The warmed, more permeable tissues respond more robustly to EMS, so you get better recruitment with less discomfort.
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5. Hi-TENS (pulsed stimulation combining low and high frequency)
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Description / physics: It’s a “TENS on steroids” — traditional low-frequency TENS pulses (e.g. 2 Hz, 5 Hz, 25 Hz) are embedded or modulated within higher frequency envelopes, allowing better penetration and deeper effects.
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Tissues targeted: Sensory nerves / nociceptors, superficial nerve endings.
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Effects & healing mechanism:
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Gate control analgesia: Low-frequency TENS modulates pain signals by activating A-beta fibers and inhibiting nociceptive influx.
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Endogenous opioid / neurochemical release: Some TENS settings stimulate release of endorphins which contribute to analgesia.
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Microcirculation boost and trophicity: The improved circulation also helps nourishment and waste removal in superficial tissues. winback.com
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Chronic pain modulation: Because of better penetration and a “richer” stimulation waveform, Hi-TENS is described as more effective in both acute and chronic pain control.
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How These Currents Speed Healing (Mechanisms & Synergies)
Putting the pieces together—here’s how using this multifrequency model advances tissue repair more efficiently than single-modality approaches:
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Thermal / Diathermic Effects + Perfusion
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The TECAR currents heat tissues internally, dilating blood vessels and boosting microcirculation, which in turn delivers oxygen, nutrients, and growth factors while flushing metabolites. winback.com
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The elevated temperature also enhances enzymatic activity, metabolic reactions, and diffusion rates across cell membranes.
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Improved Cell Membrane Transport & Ionic Exchange
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The high-frequency oscillating current can transiently modulate cell membrane permeability (or permittivity) and drive ionic exchanges. That means nutrients, glucose, amino acids, and signaling molecules move more readily. winback-academy.org
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Thus, cells like fibroblasts, endothelial cells, satellite cells (in muscle) get better substrate access and signaling environment.
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Modulated Pain / Neural Effects
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Hi-TENS modulates nociceptive afferents (pain gating, opioid release), letting tissues be loaded (mobilized) earlier with better tolerance. winback.com+3winback.com+3winback.com+3
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The thermal and mechanical (from EMS) effects reduce neural sensitization, muscle guarding, and contribute to analgesia.
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Edema / Waste Clearance
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Muscle contraction via Hi-EMS acts like an internal pump, moving interstitial fluid, lymph, and metabolites out of injured zones.
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Combined with improved circulation from heating, this ensures a “cleaner” healing environment.
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Fibroblast / Collagen Remodeling & Tissue Adaptation
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The local heating and mechanical stress (from EMS) stimulate fibroblasts to produce collagen, reorganize fibers, and lay down extra ECM in optimal alignment.
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Over time, this helps tendon, ligament, and scar tissue mature, reduce fibrosis, increase tensile strength, and restore functional elasticity.
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Synergistic Timing
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By doing heat + EMS + TENS in one session (or in rapid succession), you ensure that tissues are optimally primed (warmed, less stiff) for contraction, and that analgesia is in place so activation is tolerable.
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This synergy often means less total treatment time and better adaptation than applying each modality separately. winback.com+2winback.com+2
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Selective Depth Targeting
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Thanks to resistive vs capacitive modes, you can choose which tissues to emphasize (deep CT vs superficial soft tissue). This precision reduces collateral heating, improves comfort, and targets pathological tissues more effectively. winback.com+4winback-academy.org+4winback.com+4
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Practical Implications: How a Clinician “Chooses Currents” During a Session
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Acute inflammation / pain dominance phase:
Use more Hi-TENS (low frequencies) + moderate CET to provide analgesia, reduce guarding, and increase superficial perfusion. -
Transition / early repair phase:
Integrate CET + RET (dual mode) to warm both soft tissues and deeper connective tissues, plus Hi-EMS to begin gentle muscle activation (if tolerable). -
Remodeling / late phase:
More aggressive RET + EMS load (within tolerance) to stimulate deeper tissues (tendon, ligament) while maintaining analgesia via TENS as needed. -
Maintenance / repeat sessions:
Mix and match – sometimes just EMS (for muscle tone), sometimes TENS (for pain flares), sometimes TECAR to reset tissue metabolism. -
Application technique matters:
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Position electrodes to “traverse” the path between active and return plates such that pathological tissues lie within the current path.
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Use appropriate power settings—adjusting for tissue impedance, electrode size, and patient comfort. Winback devices have features (e.g. Power-In) to auto-adjust intensity. winback.com+2winback.com+2
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Combine with manual therapy / mobilization while currents are on to “lock in” gains.
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Current / Mode | Frequency / Type | Primary Target Tissues | Effects on Healing |
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Capacitive (CET) | ~300 kHz – 1 MHz (insulated electrode) |
Superficial, high-water tissues (muscle, fascia, fat, skin) | Increases circulation, warms tissue, boosts cell metabolism, reduces muscle tension & pain |
Resistive (RET) | ~300 kHz – 1 MHz (conductive electrode) |
Deep, high-resistance tissues (tendon, ligament, bone, joint capsules) | Targets deep structures, stimulates collagen repair, supports tendon/ligament healing |
Mixed CET + RET | Simultaneous / alternating | Both superficial and deep tissues | Combines benefits: circulation + deep repair, efficient full-layer treatment |
Hi-EMS | 1,500 – 4,000 Hz (medium-frequency) |
Motor nerves, muscle fibers | Muscle activation, improved blood & lymph flow, prevents atrophy, re-educates neuromuscular control |
Hi-TENS | Low-freq TENS pulses within high-freq envelope | Sensory nerves, nociceptors | Analgesia (pain relief), endorphin release, reduces chronic pain, enhances microcirculation |